Epidermis a meʻAneʻe
ʻO ka ʻeʻe kahi maʻi ʻeha mau loa o nā follicles lauoho a me nā kelepa sebaceous, a i kekahi manawa i manaʻo ʻia he pane physiological i loko o ke kanaka, no ka mea, kokoke nā kānaka a pau e ʻike i ka ʻeha o nā ʻano like ʻole i ko lākou ola ʻana. He mea maʻamau ia i nā kāne a me nā wahine ʻōpio, a ʻoi aku ka liʻiliʻi o nā wahine ma mua o nā kāne, akā ʻoi aku ka nui o nā makahiki ma mua o nā kāne. Ua hōʻike ʻia nā haʻawina Epidemiological e pili ana i ka 80% a i ka 90% o nā ʻōpio i loaʻa i ka haki.
Wahi a ka pathogenesis o ka huehue, ua māhele ʻia ka ʻekene i ʻekolu mau ʻāpana: ① Endogenous acne, me ka acne vulgaris, perioral dermatitis, acne aggregation, hidradenitis suppurativa, acne breakout, premenstrual acne, facial purulent skin maʻi, etc.; ② ʻAneʻe exogenous, ʻāne mechanical, ʻāneʻe tropical, ʻāneʻe urticaria, ʻeke kauwela, ʻāneʻe lā, ʻāne i hoʻohana ʻia i ka lāʻau, chloracne, cosmetic acne a me ka ʻaila hinu; ③ ka huʻi ʻana e like me ka huehue, me ka rosacea , keloid acne o ka ʻāʻī, gram-negative bacilli folliculitis, steroid acne, a me nā maʻi e pili ana i ka haki. I waena o lākou, ʻo ka acne e pili ana i ka ʻoihana hoʻonaninani ʻo ka acne vulgaris.
He maʻi pilosebaceous inflammatory mau ka ʻeʻe, a ua wehewehe maoli ʻia kona pathogenesis. Hiki ke hōʻuluʻulu ʻia nā kumu pathogenic i ʻehā mau helu: ①Ke hana nei nā kelepa sebaceous ma lalo o ka hana a nā androgens, piʻi ka huna sebum, a momona ka ʻili; ② Hoʻonui ka hoʻopili ʻana o nā keratinocytes i ka infundibulum o ka follicle lauoho, ʻo ia ka poloka o ka wehe; ③ ʻO ka propionibacterium acnes i loko o ka lauoho follicle sebaceous gland he nui ka Reproduction, decomposition o sebum; ④ kemika a me ka cellular mediators alakai i dermatitis, a laila suppuration, ka luku ana o ka lauoho follicles a me ka sebaceous glands.
Ka manawa hoʻouna: Iulai-29-2022
