Epidermis a meʻAneʻe
ʻO ka ʻeʻe kahi maʻi ʻeha mau loa o nā follicles lauoho a me nā kelepa sebaceous, a i kekahi manawa ke manaʻo ʻia he pane physiological i loko o ke kanaka, no ka mea, kokoke nā kānaka a pau e ʻike i ka ʻeha o nā ʻano like ʻole i ko lākou ola ʻana.He mea maʻamau ia i nā kāne a me nā wahine ʻōpio, a ʻoi aku ka liʻiliʻi o nā wahine ma mua o nā kāne, akā ua ʻoi aku ka makahiki ma mua o nā kāne.Ua hōʻike ʻia nā haʻawina Epidemiological ma kahi o 80% a 90% o nā ʻōpio i loaʻa i ka haki.
Wahi a ka pathogenesis o ka huehue, ua māhele ʻia ka ʻekene i ʻekolu mau ʻāpana: ① Endogenous acne, me ka acne vulgaris, perioral dermatitis, acne aggregation, hidradenitis suppurativa, acne breakout, premenstrual acne, facial purulent skin maʻi, etc.;② ʻAneʻe exogenous, ʻāne mechanical, ʻāneʻe tropical, ʻāneʻe urticaria, ʻāne kauwela, ʻāne lā, ʻāneʻe lāʻau, chloracne, ʻeneʻe cosmetic a me ka ʻaila hinu;③ ka huʻi ʻana e like me ka huehue, me ka rosacea , keloid acne o ka ʻāʻī, gram-negative bacilli folliculitis, steroid acne, a me nā maʻi e pili ana i ka haki.I waena o lākou, ʻo ka acne e pili ana i ka ʻoihana hoʻonaninani ʻo ka acne vulgaris.
He maʻi pilosebaceous inflammatory mau ka ʻeʻe, a ua wehewehe maoli ʻia kona pathogenesis.Hiki ke hōʻuluʻulu ʻia nā kumu pathogenic i ʻehā mau helu: ①Ke hana nei nā kelepa sebaceous ma lalo o ka hana a nā androgens, piʻi ka huna sebum, a momona ka ʻili;② Hoʻonui ka hoʻopili ʻana o nā keratinocytes i ka infundibulum o ka follicle lauoho, ʻo ia ka poloka o ka wehe;③ ʻO ka propionibacterium acnes i loko o ka lauoho follicle sebaceous gland he nui ka Reproduction, decomposition o sebum;④ kemika a me ka cellular mediators alakaʻi i dermatitis, a laila suppuration, ka luku o ka lauoho follicles a me ka sebaceous glands.
Ka manawa hoʻouna: Iulai-29-2022
