ʻEpidermis a me ka ʻākīne

ʻEpidermis a meʻAkika

He maʻi mumū mau ka ʻākea o nā follicle lauoho a me nā kelepa sebaceous, a i kekahi manawa ua manaʻo ʻia he pane kino i loko o ke kanaka, ʻoiai ua ʻike ka hapa nui o nā kānaka i ka ʻākea o ke koʻikoʻi like ʻole i ko lākou ola ʻana. ʻOi aku ka maʻamau i nā kāne a me nā wahine ʻōpio, a ʻoi aku ka liʻiliʻi o nā wahine ma mua o nā kāne, akā ʻoi aku ka mua o ko lākou mau makahiki ma mua o nā kāne. Ua hōʻike ʻia nā haʻawina epidemiological ma kahi o 80% a 90% o nā ʻōpio i loaʻa i ka ʻākea.
Wahi a ke ʻano o ka ʻākena, ua māhele ʻia ka ʻākena i ʻekolu mau māhele: ① ʻĀkena endogenous, me ka ʻākena vulgaris, perioral dermatitis, acne aggregation, hidradenitis suppurativa, acne breakout, premenstrual acne, facial purulent skin diseases, etc.; ② ʻĀkena exogenous, mechanical acne, tropical acne, urticarial acne, summer acne, solar acne, drug-induced acne, chloracne, cosmetic acne a me oily acne; ③ nā ʻōhū like me ka ʻākena, me ka rosacea, keloid acne o ka ʻāʻī, gram-negative bacilli folliculitis, steroid acne, a me nā syndromes e pili ana i ka ʻākena. I waena o lākou, ʻo ka ʻākena e pili ana i ke kahua hoʻonaninani ʻo ia ka ʻākena vulgaris.
He maʻi mumū mau ka ʻākena, a ua wehewehe pono ʻia kona pathogenesis. Hiki ke hōʻuluʻulu ʻia nā kumu pathogenic i ʻehā mau kiko: ①Ke hana nei nā kelepa sebaceous ma lalo o ka hana a androgens, hoʻonui ka sebum secretion, a momona ka ʻili; ②Hoʻonui ka hoʻopili ʻana o nā keratinocytes i loko o ka infundibulum o ka follicle lauoho, ʻo ia ke ālai ʻana o ka puka; ③He nui ka propionibacterium acnes i loko o ka follicle lauoho sebaceous gland Hoʻoulu hou, hoʻopau i ka sebum; ④ alakaʻi nā mea hoʻolaha kemika a me nā cellular i ka dermatitis, a laila suppuration, ka luku ʻia ʻana o nā follicle lauoho a me nā kelepa sebaceous.


Ka manawa hoʻouna: Iulai-29-2022

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